8/15/2023 0 Comments Anti mog myelitis![]() This may include IVIG and medication, such as azathioprine, myucophenolate mofetil, or rituximab. To our knowledge, this is the first documented case of anti-MOG antibody-associated TM occurring in a patient with Sjgren’s syndrome. However, some patients may experience recurrent attacks and need a long-term management plan. We describe a single case of a patient with TM associated with the presence of the autoantibody anti-myelin oligodendrocyte glycoprotein (anti-myelin oligodendrocyte protein (MOG)) who was also found to have pSS. Many individuals with anti-MOG syndrome will recover fully after their first treatment and never relapse. In rare cases, if high-dose steroids are not effective, patients may undergo intravenous immunoglobulin (IVIG) treatment. Intravenous corticosteroids are the first line of treatment. Monophasic or relapsing acute optic neuritis, myelitis, brainstem encephalitis. Treatment for acute symptoms of demyelination involves reducing inflammation. between anti MOG related demyelinating disease and other entities. Symptoms of acute disseminated encephalomyelitis may include: Recent descriptions of a demyelinating syndrome of the CNS associated with anti-MOG antibodies, referred to as MOG antibody-associated disorder (MOGAD), have been critical in differentiating these patients from those with multiple sclerosis (MS), which has divergent therapeutic and prognostic implications. Individuals with anti-MOG syndrome may experience optic neuritis, transverse myelitis, and/or acute dissemination encephalomyelitis, depending on which parts of their nervous system are affected.
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